Genetics Records Data of over 400 years is an out of the box discovery using the Baseline (coding TDMLs...) of the Paynes/DePayens (my bio dad is Reno Payne leads to Hugh De Payen) to persistently merge/expand/find/data/process coding of TDMl's... people in the dna and/or family tree to the 1600's (merging many of the 1620's people when expand/...).  The processes of the discover/discovery allows to see high dna and or family trees which can locate the high dna and/or family trees  of the very old past--the 1600's. As a unique researcher, that does not fall into the category of having a degree in genetics I ask "Is past high dna/high subset of old relations (family trees) tied to a condition of inbreeding which is a neurological disorder AUTISM. Currently there are over 120k Payne connections in my tree but millions of data collected over 8 years to show results by way of data. Mayflower/others surnames (my relationship value below) will continue to be expanded now to show their TDML's..... Fast Track (below) can show which siblings are tied to the higher cm of the specific parent but using research that involves many families with 2 or more children with and without autism can reveal more templates. Templates to compare relationship values against the "One for all tree/Veins of our Ancestors" template. Each person has their own markers for ancestors that can show more high dna/autism. Also I am hoping to find others in America that have a maiden name (if female-MF passenger) that shows a straight line to the past for other templates to be compared/merged and in the future marked (ancestors that seem likely to be a part of the high dna/autism/condition of inbreeding.

Futuristically, I see 2 dissertations combined into 1 dissertation at Arizona State University for my Doctorate. The first involves "A Genetic  Foundation of a subset of the white person's genetic make-up (1620's and beyond) of America",   which is a  good portion of America today. In using the software of Ancestry.com and the De Payen (my bio dads name is Payne leads to Hugh De Payen) family, the data is expanded since 2016 to show relationships from the Payne's to  most everyone dating down from the 1620's that were on the Mayflower. This also includes so many others dating (male to male from 1620's male person) from this past time.

The second dissertation shows the close relations (each person actually has a unique text/info which is the relationship of Paynes vs each person in my tree concerning mostly of the time period of the1620's. Passengers surnames and same surname (1620's GGF's..) male to male connections--high dna of the past. My theory involves (with other countries it could be the same) allowing for coding (TDML's -male to male, DOTDML's - daughters of each male to male from 1620's GGF... and their married name) to see the family tree/dna tree for high dna suseptibilites/crossover of others with high dna of same ethnics such as white and same subset of people marrying into the same people over 400+ years. Asking the question -- Is there a high rate of families in America with a criteria-- white and have high dna of this type and also have autism in their family? Using Ancestry.com data from my plethora of a family tree it can be proven. Is it less present when someone with ethnic or racial differences marries into this kind of genetic pool ? If this website can show TDML's, DOTDML's and married name can it be tested. Does the DNA family and placing MF surnames also show a high instance of Autism due to my high past connections? 

Fast Track - Theory to Fact - Labeling Autism

Finding the cm of each sibling of a family (each parent), locating that the siblings with autism are tied to 1 parent (due to higher cm) with the history of high subset DNA of the past by way of genetics records data -- "DePayen Baseline" research project vs ..... Note: Anyone who has a free account and paid $59 for the DNA test can see the cm for parents vs childrens cm (centimorgans). 

It would be a goal to process this type of information--my bio son (no other bio children) has a cm closest to the high dna of the past parent (more cm's to that parent). He has more cm to the mother than the father. It is my child who has autism. Since Payne is a direct line to the past--I have high dna of the past with alot MF passengers of pretty high relations/relationship title of the 1620's and before that time. I would like research to go further than helping those to find their high dna/family trees/ develop the foundation of one tree. I would like to find people that have direct lines to the surnames of the MF passengers and have families with autism. Questioning: Is there a same outcome-their bio children with autism relate more to the high dna parent of the past. Also in what order-does dna get passed on more so by the mother with the first child vs later children. Can it be more so with the father with the first child? How does this type of genetic change for time and same environment of body adjusting to same genes for births (for this kind of research). Is remission lessened by way of more births same parents or is it the other way around depending on which (female or male has the high dna) parent.

In a way Infertility and Autism have a simularity--If you have IVF or IVF Gift and it leads to a birth then you have a higher chance of having a child with autism. Questioning: Does high dna family trees/people with high dna for the past also relate to having infertility? 

Concern: After this tree is expanded (may be years to bring everyone together in one tree) allow for Hein. The DNA relationship values with 8 MFP are One: John Billington 1576 ties with my GGF Payne and they are related by way of being my 9th GGF. Two: William Bradford 1559 ties with my GGF Payne and they are related by being my bil 6th GGA. Three: James Chilton 1556 also related by by GGF Payne by being my 8th GGF. Four: Francis Cook 1584 .... by being my 10th GGF. Five: Richard Moore 1579 no descendants

Six: Myles Standish 1584 also related by GGF Payne by being my f of 8th GGA. Seven: Richard Warren 1578 also related by my GGF Payne by being my 10th GGF. Eight: William White 1591 2nd C 11x. The relationships change and become tighter/verified better/ as I continue to add wives/husbands/merge same person with same person. The other names of the MFP are: John Alden, William Brewster, Peter Browne, Edward Doty, Frances Eaton-Billington, Moses Fletcher, Edward Fuller, Stephen Hopkins, John Howland, William Mullins, Degory Priest, Thomas Rogers, Henry Samson, George Soule, John Tilley and Edward Winslow--they are all in the "One Tree for All". Currently placing all Relationship values of these last name MFP's / how they relate to me in my facebook account When working with Winslow - Hanks by way of merging same bd bd dd dd I see the dna connection of A Lincoln by way of "Friend" my mothers side but need to keep the place of A Lincoln to expand the "one for all tree" at least for now. 

 At some point I will start another tree with Abraham Lincoln on my mothers side in the correct place (it is just not develped fully) it has allowed for this " One Tree for All" to be a foundation by way of merging/verifying, others to find their family tree to the 1600's. At a later times adding another tree after the "One tree for All" is developed better then I will add Hein. It would allow for the superseding of AL to not occur(software) for more of the dna facts/relationship values to show up( such as Nine through Twentry Four which is now related through A. Lincoln. For now, we are finding answers through not just family trees and those families that can find their high dna but also through dna tree of the De Payens (One through Eight MFP  = DNA Family & One Family Tree of the Paynes). Not only looking at cm.... (see above) but also the age of the parent(s) as dna is transferred by the older male at a higher % (female age ?). Since there are hundreds of thousands of merging of same person (2 to 1) there are double parents showing up and other other mother than what the records with Ancestry are showing/what Geneanet has to offer. I have other better choices than what is offered in Ancestry.com as common answers/records. Many names that sound like ____ have been discovered and I can see they are converted to an American "like" name (and merged to one tree), this is done my our past ancestors. Labeling of all these sounded out kind of names. Because of the double parents, more mothers that brought on about when merging, a pro  needs to fine tune this tree for present/future use. A thousand people would not be enough to fine tune this tree ;) This one tree brings to light what belongs and what doesn't because of the enormous amount of merging in the 1600's-starting with many names going backwards many, many times....

RN: Aside from an enormous amount of merging the tree/expanding with extra wives/husband (placing 2 yrs younger for names I find for wives... and detailing....)  and getting the TDML's done.... I would like to see if ASU will understand/allow me to pursue  this data driven research for labeling autism. My hope is that others will find this out of the box research a foundation for charting genetics through 400+ years and find it the strong connections that I find are a test for locating autism--crossovers.  What other questions could be asked to find or see things differently. Can we find others that also have a direct line of males with the 1620's Ancestry? If male genetic information is passed on more so than female then is it a reason for the 4 to 1 ratio in autism? Are there a minute amount of children with autism that have one ethnic type of person married to a very different ethnic type of person--if so this type of data only data to find answers can be done. There is an enormous amount of Ancestry records that I have used and the foundation is my family tree/DNA tree. For about 8 years I have extended the information and merged to bring most people tied to my 1620's very well developed tree. There has been an extreme amount of  merging (same person merged with same person). The De Payen family tree/DNA tree is beyond enormous. 

Research for the discovery of this type of research of today is different from the research of discovering from the reality of the future. 

1) The foundation which is the Payne tree is what is happening in the very past (records of people born to certain families--high dna due to being in one location/marrying same type of people due to same interests (wants same ethnic/race/religion/statusover many years.

2) The processes or behaviors of these processes are detailed by way of specific coding TDML, and DOTDML's to include husband's name in Ancestry.com This is done for each MF 1620's person as they are all in my tree and below show a very close family tie. My goal is to show crossovers.

3) The outcome is Autism-condition of high dna over many crossovers (very little differences in dna/recent racial differences). A condition of inbreeding over hundreds of years.

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Discovering (same as above but...)

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1) The foundation of others (say there are 30 people in the US that have a straight line/same last maiden name of Alden...... and they perform the same kind of research I am working on/have data to show TDML's, DOTDML's and husbands name. This is linked to my very merged extensive tree.

2) The processes or TDML's.... are coded onto their Ancestry.com Family tree. I am hoping that "Autism Speaks" allows for a grant/help for this research to look at families that have autism and do not have autism with respect to the recent racial remission and look for percentages of facts ...... This is all based on facts/records not what anyone is saying.

3) If we are aware and able to label autism by way of this kind of knowledge--Autism can be understood. Autism can decline.

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Research, such an important word in the field of problem solving and I question the difference between discover and discovering. To discover--I found out (dna test) about my De Payen family until 2016--my bio dad leads straight to Hugh De Payen. It is a find/found out/discover of how I can expand/merge so many of the same families together in my Tree (Ancestry.com). Discovering involves locating the foundation of this tree and using it in many ways to see high dna of the past, locations of many births (with regards to  relationships), unexplained many  very young deaths of boys, locate high dna and/or family trees with high dna of the past, locate relations of Mayflower surnames and other popular surnames of the very old past, and allow a chance to see crossovers. It is possible to develop the corssovers.  It is a way to gain specific varied  knowledge. Coding the TDML's, DOTDML's... can help to locate a hight subset of same dna from our past. This is an enormous about of details and data entering, a possible theory to truth in seeing the unknown--Autism.

The purpose of this research data is to find and  label autism by way of genetics using the De Payen Family/DNA Tree--Ancestry.com This tree is worked on almost daily to merge most everyone in one tree --from the 1600's to about 1800's. It can show families how they relate to their GGF's over about 24 crossovers (marriages/babies since 1600's to include remarriages). If there is a high dna for this subset then Autism is explored by looking at other families that have the same--and have autism in their family. This can also be looked at in a different way--infertility. Infertility is also rising to 20% of the population. Autism is now close to 4% of 333 million in America as of the CDC rates recently published on 3/23/23.  It is concerning that a very small percentage has left the United States since the 1600's, it would be common/concerning to find out how much some of us are genetically tied to too many of the same gene (possibly same religion of groups marrying into same groups). Singapore which has the highest religious diversity shows a  rate of autism of 1 in 150,  Infertility rate almost non-existent as it does not seem to be a problem. Autism in the  US 1 in 40 children born have Autism while 1 in every female trying to conceive has infertility--20%. Autism always rising, not sure if infertility is basically the same rate--rising. 

 The Tree is enormous due to years of expanding extra wives/husband and using this tree allows for an enormous amount of merging of same names. I have all the Mayflower Passenger/others that are from the 1620's in my Family tree with Ancestry.com.  My plan is to find autism by way of placing TDML next to the last names of the MFP and veining down from male to male. My last name would have been Payne leading to Hugh de Payen my 24th GGF.  In placing information that allows us to see a concentration of the same dna of great grandfathers it will show high concentrations for same dna over an abundance of time. This same blood/dna that crosses over too many times is a theory to produce an anomaly such as a condition of inbreeding. A theory to finding a label for Autism.

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I would like to also connect DNA families by way of anyone that has a direct line (same last name all the way to the 1620's).  How do I find this person(s)?

This process of placing "TDML" for each male that has the last name of Alden..... from the 1620's  and all the daughters of the TDML's (DOTDML's) with their husband's name takes time. 

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This seems to be the perfect tree to try this very, very extensive research but there it does have the ability to show truth and it helps in creating a tree that condenses all (seems like all) information to one tree(MFP). Most of the information is from the 1400's to the 1800's. Almost all (maybe 99%) information that is available is coming from Ancestry.com -- my dad's line to Hugh De Payen. The HIgh DNA is revealed through Relationship Values (RV) which is the title of relationship between the Paynes and each MF passenger. The higher DNA/closer knit title reveals for others to see if their family (family with autism) hooks up to the same high dna--important to see if autism is related to high dna of the past. Also coding/labeling on Ancestry under Winslow's "High DNA Exponential" after the surname "Winslow". This is being added to just the  Winslow's (would like to see if others are also exponetially growing but .....) as there are so many during the 1600's with closer knit titles/RV within the Paynes/Winslows (see Winslows about 300 in the One Tree for All-Paynes. I am only listing those (prob abt 125) that are have close RV's so others can see the pattern along with my project of Veining from the 1620's MFP--looking for a high rate of autism due to high dna of same group of families staying together creating families over 400+ years. 

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Here are my Relationship Values for each MFP (this changes as there is so much merging of same people-bd/dd). I question in my "one tree for all" if any one person has only one RV-does the software program only allow for one"?

John Alden Mayflower 1598-1687 my 9th GGF (previous dna 12th GGF Paynes switch to AL RV 9th GGF-1 tree)

Isaac Allerton Mayflower 1586-1659  G of wife of husband 1st C 10X

John Billington Bullington Mayflower 1576-1630 G of wife of husband 8th GGA (Dna and of course 1 fam tree)

William Bradford Mayflower 1559-1591 bil of 6th GGA (Dna and of course 1 fam tree)

William Brewster Mayflower 1566-1644 G of wife 1st C 10X now 10th GGF (software switched to AL line of RV)

Peter Browne Mayflower 1587-1633 G of 3rd C 9X

James Chilton Mayflower 1556-1620 8th GGF (Dna and of course 1 fam tree) 

Francis Cook Cooke.. Mayflower 1584-1663 10th GGF (Dna and of course 1 fam tree)

Edward Doty Doten Mayflower 1598-1655 fil 1st C 10X

Francis Eaton - Billington Mayflower 1596-1684 fil 8th GGA

Moyses Moses Fletcher Mayflower (left his family for later trip but they did not emigrate to Plymouth, he died 1st winter)

Edward Fuller Mayflower 1575-1620 10th GGF

Stephen Hopkins Mayflower 1581-1644 10th GGU

John Howland Mayflower 1571-1621 h of 1C 11x now 11th GGF (software switched to AL line of RV)

Richard Moore More 1579-1621  no descendants (Dna and of course 1 fam tree)

William Mullins Mayflower  1572-1621 10th GGF

Degory Priest Mayflower 1582-1621 GG of wife 2nd C 8X

Thomas Rogers Mayflower 1598-1621 h of 11th GGM

Henry Samson Mayflower 1604-1684 fil 1C 9x 

George Soule Mayflower 1590-1679 fil 2nd C 9x

Myles Standish Mayflower 1584-1656 f of 8th GGA (Dna and of course 1 fam tree)

John Tilley Mayflower 1571-1621 h of 1st C 11x

Richard Warren Mayflower 1578-1628 10th GGF (Dna and of course 1 fam tree)

William White Mayflower 1591-1620 2nd C 11x (Dna and of course 1 fam tree)

Edward Winslow  Mayflower 1595-1655 h of 10th GGA

RN: Aside from families with autism being able to connect to this "One Tree for All" Hugh De Payen with MFP (RV/high dna) in the "One Tree for All" the question is "Is there a high probability connecting vs not connecting well to having Autism in the family vs not having Autism in the family due to the high dna shown in this ongoing research. Yes, I need a rewrite of this research. Here is a list of names for high dna due to the greater amount of births in the MFP's during the perioed before the 1700's (using veins of my ancestors/TDML/DOTDML/Husband's name for  families to locate their family tree/locate autism probability).

Winslow's before 1699 in my "One For All Tree"-- have not done "Veins of our Ancestors with Winslow's yet.

Categorizing close knit RV of the Paynes's (Title I see in Ancestry.com and placing "Winslow" in the search area there are about 125 that are in the period of the 1699's and before. This is high DNA/Family Tree because of the many crossovers for this group of people over 400 years (High DNA of a specific group). Because i have had to add A. Lincoln to get the "one Tree for All to Exist" and use it for the processes, some names will not have a RV that is DNA tied as it is a part of the A. Lincoln side. A Lincoln is on my mother's side (not as close as I have placed it) but it is not possible at this time to see it in a clear way. 

The "High DNA Exponential" is going after the surname of Winslow's in my One Tree for All. Why? There are over 300 Winslow's in my tree it is high dna of the past because of the numbers so the chances for crossovers would be very likely over 400 yrs.

James Winslow Chilton MFC 1556-1620 my 8th GGF by way of Nelson Payne my 2nd GGF 1848-1941, Eleanor Winslow 1677-1738 my (not close)by Harrison Payne my 3rd GGF, Mary Chilton Avery Noyce Winslow 1607-1679 7th GGA by way of Nelson Payne, John Winslow MC 1597-1674 H of 7th GGA by way of Nelson, Sarah Winslow Flight Standish Middlecott Howard 1609-abt 1683 not close by way of Stephen Payne my 8th GGF, Margaret Mary Elizabeth Hicks Winslow 1589-1666 A.Lincoln ...1c 10x, Elizabeth Winslow Clark Clarke Burton 1637-1663 not close A Lincoln, Margaret Winslow Miller 1640-1698 ...1c 10x A Lincoln, Mary Magdeline Sarah Chilton Winslow Sears Sayre Worden 1610-1687 sil 7th GGA by way of Nelson, Rebecca Winslow Thacher 1643-1683 ...1c 10x A Lincoln, Josiah Winslow 1605-1674 ...1c 10x A Lincoln, Edward Winslow 1560-1631 fil 7th GGA by way of Nelson, Elizabeth Barker Berker Winslow or Katherine Moore? w of h 10th GGA A Lincoln, Thankful Winslow Keen Crosby Crosbie 1697-1742 1c 7x A Lincoln, Mary Marcy Winslow Chilton 1628-1664 1c 8x by way of Nelson, Ruth Sargent Bourne Chipman Winslow 1642-1713 w of h 9th GGA A Lincoln, Abigail Austin Autstin Lewis Winslow Webb 1695-1747 A Lincoln, Susanna Winslow Lathan 1630-1685 1c 8x by way of Nelson, Mary Winslow Gray 1635-1665 1c 8x by way of Nelson, Edward 1635-1682/Sarah 1638-1726/Isaac /John all 1c 8x by by way of Nelson, Samuel Wilson 1641-1680 1c 8x A Lincoln, Julia C Winslow Clark 1651-1673 1c 8x by way of Nelson, Benjamin Winslow 1653-1676 1c 8x by way of Nelson, Susanna Jackson Fuller White Winslow 1593-1680 10th GGA A Lincoln, George Winslow 1691-1757 fil 1c 8x by way of Harrison Payne my 3rd GGF, Winslow Hayward 1602- not close by way of Stephen Payen 1629 8th GGF, Lydia Winslow 1693- 1c 7x A Lincoln, Snow Winslow 1698- 

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So at some point I can go into my account with Ancestry place Winslow in the search and if I can get a high dna from the exp labeling and the "Veins of our Ancestors" I could have a list of people to contact to find out if they have autism and see if there is a higher than normal rate due to the high dna processing/profiles in this chart I am trying to produce. There is the dna family info for myself and others but also the family tree, I would be working with the dna part.

RN: Since there are different " by way of Nelson, Harrison, Stephen Payne... "  in the 1699 and before of Winslow's could this be a way to see double/triple/... of the same great grandfathers within the Winslow's and more so as the more present time. I will extend also to place info for the 1700-1799 and 1800-1900 but because it is such a large amount of info--western exp may not show up. Many of the Winslow's of today could have the same GGF's coming from all angles--how to find that ? This is why the One Tree for All can be/should be expanded and used for all. Someday just a drop of blood will show your complete family tree based on yours vs theirs. No family tree needed. Does not matter about an affair or two. Kind of interesting how the future is foretelling the past.

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RN: Although they MFP relationship to the Paynes are not all close in relationship the idea back then until the 1800's would be to marry someone in you station of life. The crossovers are higher because the richer people married into the same families that were within the same station-- as they said back then. 

Finding high dna -- Alden's, Cook's are worked on now to find TDML's and DOTDML's with husbands name but in order of high dna I would add others that I see in my tree and Chilton 8th GGF, Fuller 10th GGF, Hopkin 10th GGU, Mullin 10th GGF, Rogers 11th GGF, Standish 8th GGA, Warren 10th GGF, and Winslow 10th GGA. I'm trying to expand/merge and see relationship values of each surname of MFP and others. This is inrelationship to my fathers line the Paynes--Reno Payne born 1904 to Hugh de Payen in 1100's. I would like to find women who also  have a maiden name such as above list (to label TDML's...) in my tree so I can match dna data. I would need a straight line of that particular name to the 1620's--she has had children (her grandfathers are leading to Mayflower Alden, Chilton, Cook..,  Fuller, Hopkins, Mullins, Rogers, Standish, Warren, and Winslow of the 1620's. Research--Later I can ask those in my DNA family if they are related to .... after putting Alden... in the search box at my Ancestry.com tree. At some point asking if there is austim in their family or just leaving the info here for others to look up and make a connection due to no remission of other outside relations (person married someone from another country recently). Research-- Asking Mayflower websites for help in locating this person that could have more help in identifying high dna/crossover/same grandfathers in our dna. Possibly causing an anomoloy--a type of inbreeding over possibly 24 times in 400 years. If my ancestors had relations in the 1620's they more than likely had more before that time ( some MF names are connected in my family tree before the 1620's) so it could be more than 24 crossovers of same Grandfathers. 

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To label "Autism" 1) Find high dna of MFP/other 1620... from a tree that is associated with high dna of MFP/crossovers of high dna over 400+ years using "TDML" to show lines of same grandfathers since the 1620's. 2) Find daughters of TDML's and place them on this website (not placing TDML's that do not have daughters without children or DOTDML's that do not have children--some I cannot find/develop. The merging/expanding is with everyone basically from the 15th century. To allow for this to be more precise--Ruth Alden 16th GGM 1488, Philip Cook 14th GGF 1448, John Winslow 7th GGU 1597, Richard Warren 10th GGF 1578, James Chilton 8th GGF, John Alden 12 GGF 1598, Francis Cook 10 GGF 1584. We are family and the subsets of too much of the same DNA could be a part of a condition of a neurological disability called Autism. It is a possibility and can be shown. More discoveries of how I relate to the MFP and others will be also listed as I find more information and develop/merge families/place so much information.

332 million in America 3% with Autism, that is 10 million. 4 to 1 for non hispanic whites to latino or african americans. 4 to 1 boys vs girls. Reason--Runs in families due to genetics too much of the same dna. Reason--more whites due to the original set of dna in the 1400's/1620/abt 1800's and many more times/same dna shown in my massive Ancestry.com De Payen dna/family tree. Reason--more boys than girls due to the known male gene variants are more transferable. Can we see a dna math/family tree patterns for a set of high dna which leads to a type of autism (neurological disorder). This list (The Alden's are all from the 1620's line male to male from 1620's or before that time) High DNA is explored in this research using my ever very very expansive tree. ALDEN-------------------------------------------

Adam 1781 H Anne 1808 Atkins Adkins (John) Albert 1906 Homer Maridell 1920 Yoho (Warren Harlan) Albert 1836 Washington Carrie 1866 Francena Phelps (Clarence) Alexander 1814 Hettie 1865 Kendall (Freeman H) Alexander 1794 Nancy L and/or Lucy L Honstan Huston (John Calvin) 

Alonzo 1817-Sarah 1842 Smith (Nelson) Jan 23, Eliza 1852 Tibbetts Goodrich (George), Emma Louise 1854 Crouch (Melville) Alpheus 1798-Sarah            Ann Bearce/Bearse (Horace) + Alpheus 1765-Lucinda 1798 Stoddard (?) Alvah 1820-Emily 1856 Thomas Andrew (George H) Alvin 1823-Ruth 1850 E Ahrens Ahrends Albrenas (William), Sarah 1849 A Lucas (Groonup P) Amander 1811-Amelia 1846 P Cole (Ephraim Thomas) Amasa 1775-Sarah 1806 M Dunbar (Rev. Darius Darcus) Amos 1866-Stella 1909 M A Swan (Earl M) Andrew 1684-Lydia 1727 Also married an Alden-Seth Capt  1721 will list later RN: Andrew Alden (TDML) 1684 father of daughter (Lydia) and Lydia marries a TDML (Seth Alden Capt 1721 they have 2 children--last names Wolcott and Spooner. Andrew 1721-Fear 1755 Beaumont (Isaiah) Andrew Elmer 1852- Anna 1895 Annie M Hoard McGuire (Walter H) 

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Research: Alden's also TDML's--no death date could be stillborn or very young: NN 1856 Adelbert Henry 1856 Amos Col. 1765 Arthur 1885 Augustus 1871 Charles Fredrick 1948 Chester 1870 Clarence 1896 Clarence Evan 1918 Clifford James 1904 David 1927 Dona 1906 Earl J 1885 Edward 1867 Edward 1878 Elijah 1807 Elmore Elmon 1876 Elton Dite Dike 1915 Floyd 1937 Frank 1878 Frank 1860 Fred 1854 Gene 1939 George 1824 George 1839 George 1898 George Edwin 1859 Hardin Harden 1829 Henry 1832 Henry B 1843 Henry Eben 1847 Horatio 1855 Ichabod 1806 James 1729 James 1862 James Chester 1941 John 1729 1871 1895 John Jr 1911 John 1938 1915 John Breonham 1923 John Harvey 1936 John Henry Harlow 1876 John Lincoln 1924 John Ordway 1925 John W 1933 Joseph 1801 Josiah 1724 Judah 1825 Len Leon 1824 Nathaniel 1748 Newton 1862 Richard Clarence 1871 Rinaldo 1828 Ronald Edward 1944 Samuel Sam E 1911 Seth 1776 Stanley Frank 1880 T S 1840 Timothy John 1948 Walter s 1827 Will 1880 William 1856 1872 William Croswell 1831 William F 1822 William H 1860 No names 1828 1834 1863 1940 Allen Earl 1935 Allen 1834 Robert E 1928 Henry Carolus Charles 1853 Jonathan 1862 Usaac 1821 William 1835 John 1786 Again when I follow/place TDML for the pathway male to male from 1620's Mayflower Alden I do not see the girls dying at such a young age/more girls born to TDML than boys 3 to 1 at least. If 500 TDML Alden's then 1500+ DOTDML.  Will be looking for this discrepancy or unusual find. 

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The total TDML-True Direct Male Line (share 1620's Mayflower Alden GGF and other GGF as clusters would be over 500 male people in my family tree.

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Research: Alden's Boys early deaths TDML's (male to male from 1620's Mayflower Alden Grandfather) Adoniram 1817-21 Alexander J 1834-35 Anthony 1720-20 Briggs 1848-52 Caleb 1744-47 Charles 1849-50 Charles 1878-78 George 1805-05 George 1828-29 Harry 1805-05 Harry L 1875-75 Hezekiah 1767-68 Hosea 1773-75 Isaac 1758-59 John 1719-20 John Briggs 1710-12 John 1915-1917 Jonathan 1703-04 Joseph 1693-95 Joseph Henry 1838-38 Judah 1790-92 Leonard Lewis 1840-42 Orlando Fuller 1839-40 Thomas 1701-01 Webster Daniel 1838-39 Alex 1850-51 

Notes: Wish I could get my nephew or someone in the family to write " The Veins of our Ancestors" The TDML's according to the relationship value between the Payne's and various different surnames of the Mayflower passengers to include how the hundred of Captains fit in. It is kind of a history book unfolding. If I can find someone that is my dna relation by way of 10th GGF due to the strange last name--can I find others that are in my dna family that are a straight true direct male line to some of the 1620 grandfathers. I am clearly tied to this Mayflower population. 

RN: The merging of same people within my tree same bd dd leads to a change of John Basse (changing all Basse to Bass) from 9th GGF to a GGU and Samuel Bass 1600-  becomes the 9th GGF. Started the TDML's for this line. I will list the passed on infants/very young toddler (TDML's of Alden's) 10% of the Alden's TDML's above along with those that do not have dd for the TDML's of the Alden's. This may be a shortcut and somethings that is duable for getting quicker answers or seeing a problem as I will also be looking at deaths for infants for DOTDML's of Aldens. The "Word" program will be used to create attachments for this large group of info (TDML and DOTDML) later for many surnames.

RN: The TDML's for the Alden's are about 500 people they are labled after the last name TDML in my Ancestry.com Payne/Depayen family tree. Around 10% of the boys have died (of the TDML's for the Alden's) at an infant age or before the age of 2. Girls are 3:1 in that girls are likely to be born for all TDML's in the Alden's. There are 81 without a death date so the 10% deaths of the small boys could be higher. I do not see any girl death dates at an early age and I have been expanding the DOTDML's. This is kind of a red flag. When I detail the DOTDML's and show the husband's name for posssible tie to more autism due to the high dna for families,I may find some girls that have died early on but nothing yet. Kind of strange findings.  After the DOTDML's I will add Capt John Bass 1630-1716 since he is my 9th GGF ( for finding TDML's). 

RN: Although it would be easy to cut and paste or place an attachment for the Alden's and Cook.... TDML's I will not be doing this but keeping a notebook of this(print out) and instead placing the DOTDML's showing the husbands name. Why? Because the whole process of finding high dna of MFP/Others would be from the women who have the high dna and have children--I want to show/have others see the information and see that it ties to the--belief too much dna of the same can cause a condition of inbreeding that leads to a neurological disorder. AUTISM. I have been expanding the DOTDML's and will probably start to list them in a few weeks for Alden and Cook... I would still need to develop all the other names that the Payne's/ DePayens are closely related using Project RV of what is in my tree. Yes, I need time.

RN: Approximately 350 TDML's for Alden's just in my family tree and about 340 TDML's for Cook.. All TDML's share the 1620 Mayflower passenger but depending on how it is veined they can share many of the same GGF. What is more important the DOTDML's as they carry the high DNA of the past and it is over and over with the same dna. I will have to add the husbands later. Still adding TDML's to the tree (most extensive tree in America). So much merging of same bd dd/person for a truer tree. Noticing that young boys are not living past 1 not seeing seeing this in girls or TDML's. TDML's and DOTDML's have more high dna than anyone else. Anyone a DOTDML living today? Like myself? Also more girls born than boys of TDML's. Should be a research on the varied female wives and how they end up having more boys. Asking again if you have too much Mayflower DNA then Autism? Asking if I have about 700 TDML just in Alden and Cook then how many DOTDML's do you think ..... More than 10% MF ancestry not if we share GGF history. DOTDML's for today need to be researched.  All I can do is post and hope that families with autism will see a genetic path and this will be researched again for facts. I will extend other surnames names, this take time....................

Alongside with history, many of us have tried to make problems a thing of the past. Research involving this type of research is extremely problematic because it involves billions of details and relies on the De Payen Family tree for its data. The most important data to find autism through genes would be by the way of the DOTDML (daughter of the true direct male lines of the MFP/Others at that time). I have created a vein(s) from the 1620's Mayflower Aldens (1 grandfather) down to the many fathers and sons. It is the daughter and their husbands names that are important in finding a strong pattern of autism due to the strong dna of the past. My belief is that the genetic problem lies with too much of the same dna over about 550yrs (at least with tree). In research I ask "are the DOTDML's high with autism in their families due to little or no recent racial genetic differences". It has been years of developing the tree and I can only offer the TDML of Alden and Cook for right now. I have a background of working with extreme high data and receiving rewards but this is very challenging for one person. I will start plugging in my TDML's today. This (TDML) is the 1620's grandfather to father to son to son ... down to say the early 1900's. I am suspecting there are many thousand of TDML's in my tree. There is autism in the family (Payne/DePayen-father's side) and also with my mother's side (Clark's and Roger's). 

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Within the time frame of 3 months I have found that using the Payne / De Payen Family tree shows history over 600yrs with many TDML's male to male (most genetic info sent from descendant to ascendant or grandfather to grandfather) I have placed the "TDML" so far with the Cooks.... and Aldens in hopes that others (esp those that have autism in their families) will expand their family tree and show that many same lines of grandfathers/surnames within 600 yrs of baby making/having children with the same genes. Many started to have families even before their 20's. So far I have a great tree to show the TDML's as there would be more to come as I keep working on this. My 16th GGM is Ruth Alden from  1488. My14th GGF is Philip Cook from 1448. Here is the Discovery of the Cook's... " The TDML's :                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                     Here is the discovery of the Alden's TDML's                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                                    RN: If the ratio exist of the 4 to 1 male to female autism rate and 6 to 1 male to female autism rate in New Jersey and no one can give and explanation then is it because of the line of Grandfathers of each surname causing a type of inbreeding over 600yrs. Is it genetics over the course of time with some locations being hit more with this kind of theory to fact ?

In working to merge/expand all MFP to one tree and find autism has been difficult but the Cook's are worked on because they are going to be detailed. All Cooke, Cocke, Coke has been changed to Cook for the first surname so the software picks it up. Since New Jersey is the state that has 6 to 1 males with autism and the TDML has been very much veined down from my tree it makes sense to look for the cause (genetics) for autism through the Cook's. Most Cook's are coming from the Novia Scotia area and staying in New Jersey. Most of my old research notes go from exceptionally large ideas/information/projects to downsize/find a better way to see the effect. Mostly this is done because there is just one person looking at this/trying to find research from my own thoughts--Tree (A) Processes (B) and Effect-Autism (C). Upcoming data on the many many Cooks and Philip Cook 1448-1503 14th GGF by way of my Payne family. I only will provide round one of the DOTDML (husbands name) for Cook's... but in the future my guess is that there will be more families with Autism ties to the Cook's due to the theory to fact that our genes play a part in "why" we have autism.

There is a tremendous amount of information/attachment to work on but need to add Project 5 after "The List". It is taking the DOTDML and also looking for high dna like project 1 using the Payne RV. It is to get a clearer concept/high dna of ties to their particular Mayflower TDML to the connection of other MF marriages such as I did with seeing Alden's being a part of the Payne's earlier than the 1600's. 

Futuristically, finding more high dna RV-relationship values before the 1600's and finding more crossovers after the 1800's would provide more information. As the website is continuing to get attachments added for the projects 1-4 along with "The List" maybe more families of autism will see the patterns and develop their family/dna family trees. Somewhere in America there would be daughters of the TDML's (Mayflower and "Others") still alive today--they can also go through the same processes/projects to come up with their unique set of data. Someday it will be added. I think within 2 years this will look different and be more relevant if not for showing autism--a way of too much of the same dna over 400yrs then for other reasons. This website mayflowerdadepayenautismresearch.com is a very much expanded way of genetic research that is different. For now, it is read bottom up as it is only 3 months old and pretty raw. The data is also mostly from old history to the about 1800's. 

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This section is called "The List" and will be used to give a sample of the Aldens project 1 and Cook's project 2 --attachment also given later. Project 1: Alden's--DOTDML surnames are:__________________________________________________________________________ Project 2: Cook's DOTDML surnames are:____________________________________________________________________________________________________

Never ending: For each family that can hook up to these names it also needs to follow down to the date of birth of a person with autism. I'm happy to be able to find this core. Maybe it will help 

Reflect: It is funny that in taking a class at ASU they asked what is your favorite movie--it was Lorenzo's Oil. He also exercised the "B" or processes to change the condition of the diagnosis. I guess there is the "disease, condition and those that want to research--the exercise to change the condition of the disease.

I would hope to find answers in New Jersey with Autism Speaks/with families that have a high ratio of male to female autism. I'm not sure how? But I can list the surnames--getting a little closer to find theory to truth. A possibility.

RN: If I can gather the daugters of project 1 high dna for Baseline and keep a pool of surnames for Project 1 and compare to Project 2 DOTDML surnames what do I see. If I can gather both pool names of both Projects then compare to families that have autism what do I see. Do I see remission with others that have married with varied genetic backgrounds ? Surnames RV =husbands name of say Payne to high DNA for Alden's  and surnames for = husbands name for the DOTDML.

RN: It would be very interesting to label the DOTDML of each TDML and vein if they are married to another man who is also a TDML--will this happen and happen more than once ? Very interesting  but  not stupid

RN: At some point DOTDML will be placed for every daughter of the TDML-useful in finding more accurate genetic data for seeing patterns in order to show common whole groups of say possible high rates of Autism. This is Project 4 . Looking at the data of Project 1-4 and how we tie strong commonalities of what it happening with _________ within certain families.

RN: When getting results from Project 2 (finding veins to all TDML's from say Cook's) I find that the 1 person for Cook's DNA close/TDML would be my 12th GGF Anthony Cooke Knight of the Bath Scholar Sheriff of Essex Hertford Tutor to King Edward VI 1504-June 11 1576-Hammonds are inbetween the Cooke and Payne TDML. RV is with 2nd GGF HP 1799-1866. 13th and 14th GGF --John Cook Knight of the Bath 1474-Oct 10 1516 and 14th GGF Philip Cooke Knight of the Bath General 1448- Dec 7 1503 same ties and between also Hammond. They are TDML's and below are Cook  9th cousins by way of NP1848-1941 1st GGF Arthur 1895-1993 Charles W 1884-    Fred W 1898-  George G 1896- Gilbert E 1894-  Russel L 1886- Sherman A 1892- Smyth Sonyth Sith Hatfield 1885-Aug 23 1921 Walter W 1902- William H 1884- 

There are also GGU TDML's Jacob Cooke 1618-Dec 17 1675 9th GGU by way of Hp 1799-1866 my 2nd GGF, Richard 1578- 10th GGU same, William 1582- 10th GGU same way.

Currently working to place Cook then Cooke or Cock or Coke or Couke for each of these type of surnames in my family tree so merging same with same occurs and TDML is accurate for veining process. Also to vein my DNA BASELINE TDML's for just "TDML" from my Payne family --need a couple weeks for this as I am also working to replace Paine to Payne then Paine to .....

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RN: When getting results from Project 1 using Alden's and any first name Alden's I find that the closest  1 person for Alden's DNA close/TDML would be my 15th GGF RV line is as follows: John Henry Alden 1510-1567, Richd Alden de Scrooby 1531-98 George Joseph Alden Aldane 1548-1620, John Mayflower Alden 1598-1687 Ruth Alden Aulden Bass 1637-1674 Samuel Bass 1660-1751 Samuel Bass Blodget Blogget 1700-1768 Hannah Bass Blodget Blogget Paine 1628-1688 John Paine 1658 Bass/Blodgett/Bloggett added to my list of surnames MFP as another added surname/important for gene duplication finding Autism.

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 RN: Not techy but after getting this 1) example "Alden" for Project 1 explained better below and 2) example "Cook" for Project 2 explained better below I will be placing pdf uploads into Wix for surnames since it is a lot of information--this will be for Project 1,2,3. As all the old notes are not worked on. The old notes brought me to this research that seems to be a good place to be for finding...

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RN: There is another Project 3 but it is being done off the record/website it includes a notebook where the NP1535 changes to an earlier RV on Project 1 (found while working with Brewster's in my family tree) this is probably due to the fact of many merges of same person with same person of b/dd. It is important (Project 3) because I see that the result of Project 1 leads to my highest DNA relative surname of the first researched from Project one which is _______________________________________________________________ Alden by way of ______________________________________________ so this notebook will have the Brewsters showing changes of RV dates (2) (some change from NP1535 to JP1658 for example) and so far one name of the Alden and one name of the Cook's in my family tree. Why?-Because direct lines of whoever with an earlier crossover may be more important (than Baseline TDML and/or TDML) in looking at the "why" in Autism--people may have same grandfathers that are strong in their dna. Such as a person having  the same 14th GGF of a cook with the same direct line of a 7th GGF Cook. The name of the Alden and also the one found ___________________________Cook by doing Project 2 is in the notebook and more will come--this is alot of work. They are basically strong DNA for Payne but past strong DNA for another surname such as above Alden's and Cook's. This notebook of the one name of Alden for me and also Cook is a crossover of old 1620's strong ties  to the (being aware) of more current ties of another's family's tree--family with autism. It is another way to use the Baseline of the Paynes but with a strong crossover. To find... To possibly find... There really are so many types of crossovers but male to male would be something I can work with. It has been difficult to write this as a common story its more of research notes to find/get more answers into math/genetics.

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Fast Track? Instead of looking      at each MFP surname and various first names by way of RV-Relationship value/Ancestry.com--place surname in my family (male to male) tree surname search see date of marriage  relationship/relationship label I will be working with Project 2. Project 2 may be more important for exploring my DNA family, this may lead to more of an accurate answer for finding autism( a type of autism-there are over a dozen).  I want to start my list of the DNA family to get the core of the information such as  asking questions of autism,recent racial genetic differences and finding each of their direct lines (the TDML's) for the MFP. I already know I have a high dna set for the MFP's. These are the volunteers I would really want to find answers. Project 2-- I Used the Cooke's, so starting with the one MFP Cooke   "Mayflower Cook" it was a little difficult to descend from the one person 1620's Cooke's in my family tree, keep track of the various veining, label eash male to male "TDML", expand to the max of information and merge same person to same person in my massive tree. Project 2 is more important than Project 1 as I will be linking the TDML to the DNA family of mine with DNA family surnames of the MFP, this will be to find ...  I am unsure if verifying/seeing a clue/is the answer but DNA does tell the truth. I'm a little unsure if this is the best way to go to get faster results. Just having a website to show information may help to gain insight into what I see. The hope of finding Autism. I have all 1620's MFP in my family tree so I will be placing "TDML" next to  their surname area(in Ancestry.com). This is for male to male/son to son True Direct Male Line next to   many descendants of of the one 1620's MFP. This is not just the Cooke's as I have done but all/each surname.  Project 1 is done below with "Alden's" (see "DATA"). I will also finish Payne's labeling in surname area "BASELINE TDML". I doubt there will be any other Baseline TDML's because most of my data is from earlier times. Ancestry does not carry information on the living/for privacy reasons. There are probably at least 300 for Cookes of TDML IN JUST MY TREE.  The question to ask is: Is this a formula to see the genes in autism--too much of the same dna/too much of the same male to male variants has a role in the  4:1 ratio of male  leading to Autism?

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I will be collecting information from my DNA family with and without common ancestors which is quite a bit of people. I will be trying to find their TDML or MFP line through surnames in my specific family tree/dna tree. This is to record what I can ant look for answers by way of dna--a more trueful way to find correct answers into Autism. Later, I will try to continue the 2 Projects alongside working with my dna family/question of autism/TDML to MFP and question of recent racial differences. I should have some answers.

History behind This Almost Three Month Old Research-mayflowerdadepayenautismresearch.com Deborah West This research involves working with many times/places/people. In research, by example such as behavioral research—Skinner/Bijou the “Antecedent” is the __________ of whatever is happening before the behavior. In my research the Antecedent is the massive Family Tree of the De Payens. This tree is the baseline used to find Autism by way of many times/many places/many people. In particular it is with the people landing from the Mayflower ship in the1620’s-- the Mayflower Passengers/close family members which is also the Paynes. The family tree is also my DNA family tree (biological father’s last name is Payne and it is direct male to male up to Hugh De Payen). This tree is the most enormous/detailed/merged of all tree’s with Ancestry.com to include a wealth of Mayflower Passengers and family surnames. It has an enormous amount of information—people from mostly the 1600’s and 1700’s. The merging of same names/birthday/death dates of various people allows the tree to become a truer tree or a “One Tree for all”. This work is done by myself--Deborah West as The Payne Tree “momdeborah52” has been expanded/detailed/merged for many years. It really is my Trademark for research both with the family tree to provide information and also the DNA family tree to research and possibly use for verification into the “Why” of Autism. It is with Ancestry.com. The two processes used within the Ancestry.com family tree is done to allow us to become more aware that genes play an enormous part in finding the “Why” –why we have autism. The Trademark tree is the best one to use and may be the only one. In reading below, the “Data” and “TDML True Direct Male Line” will work off of the specific people/time/locations. This Trademark or De Payen Family tree is in place of Skinners same idea of “A B C”. The Antecedent is a way to explain the “what is happening before the interaction”, the “inter-action” is replaced by Skinner/Bijou’s “Behavior”). I have done this to use a popular reference/skill/methodology, it is unique in that I see also a Trademark, Copyright and possible Patent. This research has started the process of relationship data/relationship value (RV) by comparison as Ancestry shows relationships. Surname and any first name in the family tree can provide the relationships and then record the direct line. It is done by way of the “Data” section and “TDML” section. In much ways, the “Data” and “TDML” information is the inter-action which has the same kind of thought process as Skinner/Bijou’s “Behavior” . The Behavior is replaced by processing a type of research to see how the family tree can be made more aware of its role/how it plays its role—Baseline up against each surname. This “Data” is a way to see the high dna of a surname or closest relative but also provides an active way to see various information over time (marriages, many same set surnames, true male to male direct line MFP crossovers with other TDML crossovers over time. This process will show crossovers and duplications as some same set DNA families are in certain areas of the US. This alteration of looking at information various processes can provide a way to see things differently. The norm “behavior” is changed by way of seeing the problem verifying Autism “the Consequence” Skinner/Bijou. It is my belief that genes can tell a story. The problem is not the behavior but in my term it is the change in physical anatomy. My belief is that there is a condition over 400yrs of mating with the same dna we create this problem. The problem is the “Why”, why we have this consequence of Autism. It is a goal/project to find is the highest relationship just from the family tree and all the records I have been inputting for many years—Ancestry.com. This is accomplished by a kind of algebra-relationship value-one baseline information name vs another surname by way of Ancestry.com This “Data” is pretty detailed as it looks at each relationship for each surname of the MFP but also others that are very strong in marriages at the time of the 1600’s. The second goal is to find all male to male descendants from the main Mayflower Passenger and place TDML—True Direct Male Line so I can use this in my process to find answers in the coming months. Both projects allow for more time spending to expand the tree and find more merging of same data to one data/one person. This is a great tree to work with as it has all the Mayflower passengers in the tree. DNA variants are carried much more with the male to male family line. Going over the course of 400+years is possible. This Research has many Questions/Notes/Hypothetic views Is there one landing of the set of dna in the 1620’ and another in the late 1700’s in Western Expansion times, can this be seen by the Payne’s by way of marriages and locations—little chance to find a mate outside of this group—Caucasion/Christians. Can showing a high dna for each of my surnames (plus) along with the TDML provide answers to most people (my results would in turn be their results). This is true for goal one but may be worked through as a template for the second goal. When looking at most of the people that have extensive family tree’s and comparing Autism/low or no varied recent genetic differences do we see high MFP dna for a specific group/autism vs low or no MFP dna for a specific group/no autism. I have not done this but it can be done.

It is a known fact that males carry the most variants to their male children and older fathers also send more dna variants than younger fathers—mine was 57 years old. I have heard twins can carry different variants. If there is a connection to the “Data” –Goal one and TDML –Goal two then future questions should be asked about can this condition cause infertility, ADHD, depression. Can the physical anomaly of the brain be like a poor immune system in handling the shots at such a young age. In autopsies done on people that have died (lifespan is shorter) and have autism there is proof that a portion of the brain cells are problematic.  This susceptibility can be a reason for the change into autism, as many parents have seen this change. Can we prove this if we know how we can see autism/what it is. If there is a connection to the “Data” and “TDML” what causes the more severe types of autism. Why are there over a dozen types of autism. Do certain people carry the same over and over same dna from long ago—other countries. Is remission of this same dna and another race/country a ticket to ride—no autism for a generation—just to start up again.  My very strong ties are with the Payne’s, Cooke’s (found with TDML process), Alden’s(found with RV process) and I have so much more to find…. In process 1 everything is found through my father’s side, very few with the Rogers, Clarks on my mother’s side. Since my grandfather married a second time into the same family as his brother then I added the first wife Mary Jane Harris and added Lincoln. This is done basically because I did not know I was going start this research, it was to purely  expand my tree. It does expand it and merge it as there are so many more record with the Lincolns. I needed to expand my tree and it was already done by the time I started exploring with people/time/and ways to process this research. My uncle has a Pulitzer Prize for his book “The Dred Scott Case” Don E Fehrenbacher. It was to find out if I can get a tree going since my mom’s dad and his brother wife would already be a part of the same dna family. Can I pull up each TDML for each surname and place in the beginning part of this research, Can I place the information on the “Data” to show a specific time and place of marriages for various surnames, Can I see correlation from “Data” of two TDML Paynes and two TDML whatever two same surnames over time. Creating various list of times/locations, specific TDML Paynes and high relationship values as a template with more to be researched.

Note: TDML are Father to son births that do not have a current connecting time for the recent present while Baseline TDML does have a current connecting time for the present. One example is my "Payne" tree. There will be many to label as TDML in my tree but for privacy this is an area for a future private agency to keep track of--well it is/will be in Ancestry.com

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